Mechanisms of Gastrointestinal Growth and Transformation

胃肠道生长和转化的机制

• 批准号: 8088362
• 负责人: JUANITA L. MERCHANT
• 金额: $ 36.89万
• 依托单位: UNIVERSITY OF MICHIGAN AT ANN ARBOR
• 依托单位国家: 美国
• 财政年份: 2010
• 资助国家: 美国
• 起止时间: 2010-08-01 至 2011-07-31
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/8088362
• 关键词: Acute; Affect; Alleles; Anti-Inflammatory Agents; Anti-inflammatory; Apoptosis; Apoptotic; Ataxia Telangiectasia; Azoxymethane; Binding; Binding Proteins; Blood Circulation; Butyrates; Cell Count; Cell Cycle Arrest; Cell Differentiation process; Cell Line; Cells; Colitis; Colon; Complex; Cyclin-Dependent Kinases; DNA; DNA Damage; DNA Double Strand Break; DNA Repair; Dextran Sulfate; Diarrhea; EP300 gene; Elements; Enterochromaffin Cells; Epidermal Growth Factor; Epithelial; Epithelial Cells; Exhibits; Expression Library; Fluids and Secretions; Funding; Gastrins; Gastrointestinal tract structure; Gene Targeting; Genetic; Genotoxic Stress; Genus Cola; Germ-Free; Growth; Histone Deacetylase Inhibitor; Histone deacetylase inhibition; Housing; Immune response; Infection; Inflammation; Injury; Intestines; Irritants; Knockout Mice; Libraries; Link; MAPK8 gene; Maintenance; Mediating; Mediator of activation protein; Microarray Analysis; Mitochondria; Morbidity - disease rate; Mucous Membrane; Mus; Mutate; Neoplastic Cell Transformation; Neuroendocrine Cell; Nuclear; Pathway interactions; Patients; Phenotype; Phosphorylation; Pituitary Gland; Plasma; Play; Polyps; Proteins; Rattus; Recruitment Activity; Reporting; Role; Screening procedure; Serine; Serotonin; Signal Induction; Signal Pathway; Signal Transduction; Small Intestines; Source; Stem cells; TP53 gene; Testing; Trichostatin A; Tumor Suppressor Proteins; Volatile Fatty Acids; Zinc Fingers; ataxia telangiectasia mutated protein; c-Myc Staining Method; cDNA Expression; cell growth; cell motility; chromatin remodeling; clinical effect; gastrointestinal; histone acetyltransferase; in vivo; microbial; mortality; notch protein; olfactomedin; oncoprotein p21; prevent; promoter; protein complex; protein expression; protein protein interaction; public health relevance; receptor; response; transcription factor