Genetics and Haloperidol Response

遗传学和氟哌啶醇反应

• 批准号: 7783812
• 负责人: ROBERT J. HITZEMANN
• 金额: --
• 依托单位: PORTLAND VA MEDICAL CENTER
• 依托单位国家: 美国
• 财政年份: 2009
• 资助国家: 美国
• 起止时间: 2009-04-01 至 2013-06-30
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/7783812
• 关键词: Address; Adverse effects; Affect; Agitation; Alternative Splicing; Animals; Antipsychotic Agents; Area; Arrhythmia; Basal Ganglia; Behavior; Brain; Breeding; Catalepsy; Chromosomes, Human, Pair 1; Chronic; Clinical; Clozapine; Code; Congenic Strain; Consensus; Corpus striatum structure; DNA; DNA Sequence; Data; Databases; Delusions; Dementia; Development; Diabetes Mellitus; Disease; Dissection; Dopamine; Drug Design; Elderly; Evaluation; Event; Evolution; Funding; Gene Expression; Gene Expression Profile; Genes; Genetic; Genetic Polymorphism; Genetic Structures; Genetic Variation; Genotype; Grant; Hallucinations; Haloperidol; Hand; Haplotypes; Huntington Disease; Inbred Strain; Individual; Maps; Masks; Mediating; Mental Health; Messenger RNA; Methods; Modeling; Mouse Strains; Movement; Movement Disorders; Mus; Network-based; Nucleic Acid Regulatory Sequences; Parkinson Disease; Parkinsonian Disorders; Pharmacotherapy; Phenotype; Population; Psychotic Disorders; Quantitative Trait Loci; RNA Interference; Regimen; Regulation; Research; Research Support; Resistance; Resolution; Role; Scientist; Sequence Analysis; Single Nucleotide Polymorphism; Single Nucleotide Polymorphism Map; Structure; Symptoms; System; Testing; Therapeutic Index; Transcript; Transfer RNA; Transgenic Animals; Veterans; Viral; Weight Gain; Work; atypical antipsychotic; functional genomics; gene function; improved; mouse model; nervous system disorder; public health relevance; receptor; response; trait; treatment strategy; vector