ROLE OF CD8+ CELLS ON SIVMAC1A11 REPLICATION

CD8 细胞对 SIVMAC1A11 复制的作用

• 批准号: 7562205
• 负责人: KOEN K VAN ROMPAY
• 金额: $ 18.16万
• 依托单位: UNIVERSITY OF CALIFORNIA AT DAVIS
• 依托单位国家: 美国
• 财政年份: 2007
• 资助国家: 美国
• 起止时间: 2007-05-01 至 2008-04-30
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/7562205
• 关键词: CD8-Positive T-Lymphocytes; Computer Retrieval of Information on Scientific Projects Database; Funding; Grant; Immune response; Institution; Research; Research Personnel; Resources; Role; Source; Subfamily lentivirinae; United States National Institutes of Health

This subproject is one of many research subprojects utilizing the resources provided by a Center grant funded by NIH/NCRR. The subproject and investigator (PI) may have received primary funding from another NIH source, and thus could be represented in other CRISP entries. The institution listed is for the Center, which is not necessarily the institution for the investigator. Objective: To get a better undertanding of the immune responses that control nonpathogenic lentivirus replication.

该副本是利用众多研究子项目之一 由NIH/NCRR资助的中心赠款提供的资源。子弹和 调查员（PI）可能已经从其他NIH来源获得了主要资金， 因此可以在其他清晰的条目中代表。列出的机构是 对于中心，这不一定是调查员的机构。 目的：更好地承担控制非对慢病毒复制的免疫反应。