POSTNATAL REMODELING IN DIST AIRWAY OF INFANT MONKEYS EXP TO OZONE & ALLERGEN

暴露于臭氧的幼猴的产后呼吸道重塑

• 批准号: 7562152
• 负责人: CHARLES George PLOPPER
• 金额: $ 5.74万
• 依托单位: UNIVERSITY OF CALIFORNIA AT DAVIS
• 依托单位国家: 美国
• 财政年份: 2007
• 资助国家: 美国
• 起止时间: 2007-05-01 至 2008-04-30
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/7562152
• 关键词: Address; Air Pollutants; Allergens; Asthma; Bronchoconstriction; Childhood; Computer Retrieval of Information on Scientific Projects Database; Exposure to; Funding; Grant; Infant; Institution; Lung diseases; Monkeys; Nature; Oxidants; Ozone; Research; Research Personnel; Resources; Source; United States National Institutes of Health; lung development; postnatal; respiratory smooth muscle

This subproject is one of many research subprojects utilizing the resources provided by a Center grant funded by NIH/NCRR. The subproject and investigator (PI) may have received primary funding from another NIH source, and thus could be represented in other CRISP entries. The institution listed is for the Center, which is not necessarily the institution for the investigator. Objective: Airway smooth muscle has been implicated in the excessive bronchoconstriction of asthma. The present study addressed the question of whether the episodic nature of urban exposure to oxidant air pollutants contributes to the rise in childhood respiratory diseases by altering postnatal lung development.

该子项目是利用该技术的众多研究子项目之一 资源由 NIH/NCRR 资助的中心拨款提供。子项目及 研究者 (PI) 可能已从 NIH 的另一个来源获得主要资金， 因此可以在其他 CRISP 条目中表示。列出的机构是 对于中心来说，它不一定是研究者的机构。 目的：气道平滑肌与哮喘的支气管过度收缩有关。本研究解决了城市暴露于氧化剂空气污染物的偶发性质是否通过改变产后肺部发育而导致儿童呼吸道疾病增加的问题。