MOLECULAR DEFECTS OF INSULIN SIGNALING IN PCOS

PCOS 中胰岛素信号传导的分子缺陷

• 批准号: 7606134
• 负责人: Ricardo Azziz
• 金额: $ 0.47万
• 依托单位: LUNDQUIST INSTITUTE FOR BIOMEDICAL INNOVATION AT HARBOR-UCLA MEDICAL CENTER
• 依托单位国家: 美国
• 财政年份: 2007
• 资助国家: 美国
• 起止时间: 2007-02-01 至 2007-11-30
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/7606134
• 关键词: Acute; Adrenal Glands; Androgens; Computer Retrieval of Information on Scientific Projects Database; Corticotropin; Defect; Family; Funding; Grant; Inherited; Institution; Molecular; Patients; Polycystic Ovary Syndrome; Research; Research Personnel; Resources; Source; United States National Institutes of Health; insulin signaling; response

This subproject is one of many research subprojects utilizing the resources provided by a Center grant funded by NIH/NCRR. The subproject and investigator (PI) may have received primary funding from another NIH source, and thus could be represented in other CRISP entries. The institution listed is for the Center, which is not necessarily the institution for the investigator. The study aims to determine whether Adrenal Androgen and Androgen response to acute ACTH stimulation is inherited within families of patients with Polycystic Ovary Syndrome.

该子项目是利用该技术的众多研究子项目之一 资源由 NIH/NCRR 资助的中心拨款提供。子项目及 研究者 (PI) 可能已从 NIH 的另一个来源获得主要资金， 因此可以在其他 CRISP 条目中表示。列出的机构是 中心，不一定是研究者的机构。 该研究旨在确定肾上腺雄激素和雄激素对急性 ACTH 刺激的反应是否在多囊卵巢综合症患者的家族中遗传。