Oxidative Injury to Gastric Epithelial Cells by H. pylori

幽门螺杆菌对胃上皮细胞的氧化损伤

• 批准号: 8332998
• 负责人: Sheila E. Crowe
• 金额: $ 32.18万
• 依托单位: UNIVERSITY OF CALIFORNIA, SAN DIEGO
• 依托单位国家: 美国
• 财政年份: 2001
• 资助国家: 美国
• 起止时间: 2001-09-21 至 2013-04-30
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/8332998
• 关键词: Address; Adenocarcinoma; Affect; Amino Acids; Apoptosis; Area; Bacteria; Base Excision Repairs; Binding; Biochemical; Biological; Biological Assay; Calcium; Cancer Etiology; Cell Cycle; Cell Death; Cell Line; Cell Proliferation; Cell Survival; Cell physiology; Cells; Cessation of life; Chronic; Cysteine; DNA Binding; DNA Repair; Data; Development; Disease; Enzymes; Epithelial; Epithelial Cells; Epithelium; Equilibrium; Event; Family; Funding; Gastric lymphoma; Gastric mucosa; Gastritis; Gene Expression; Gene Expression Regulation; Generations; Genes; Genetic Transcription; Goals; Growth; Health; Helicobacter Infections; Helicobacter pylori; Human; Human Cell Line; Immune response; In Vitro; Individual; Infection; Inflammation; Inflammation Mediators; Inflammatory; Injury; Integration Host Factors; Interleukin-8; Interphase Cell; Knowledge; Laboratories; Lead; Lesion; Leukocytes; Life; Malignant Neoplasms; Mammalian Cell; Measurement; Messenger RNA; Molecular; Mucous Membrane; NF-kappa B; Neoplasms; Nuclear Translocation; Oxidation-Reduction; Oxidative Stress; Pathogenesis; Pathway interactions; Patients; Peptic Ulcer; Phagocytes; Play; Prevention strategy; Process; Proliferating; Promoter Regions; Property; Proteins; Public Health; Reactive Oxygen Species; Recruitment Activity; Reporter; Research; Response Elements; Role; Signal Pathway; Signal Transduction; Stomach; Stomach Carcinoma; Stomach Diseases; Stress; Testing; Transcription Factor AP-1; Transcriptional Activation; Translating; Ulcer; Work; activating transcription factor; base; cell growth; cell injury; chemokine; cytokine; endonuclease; gene function; human tissue; hypoxia inducible factor 1; improved; in vivo; interest; malignant stomach neoplasm; member; protein expression; repaired; response; transcription factor