AMPA RECEPTORS AND DENDRITE BIOGENESIS

AMPA 受体和树突生物发生

• 批准号: 8167395
• 负责人: FIONA M INGLIS
• 金额: $ 6.39万
• 依托单位: LSU HEALTH SCIENCES CENTER
• 依托单位国家: 美国
• 财政年份: 2010
• 资助国家: 美国
• 起止时间: 2010-02-01 至 2011-01-31
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/8167395
• 关键词: Antipsychotic Agents; Biogenesis; Computer Retrieval of Information on Scientific Projects Database; Dendrites; Funding; Grant; Institution; Morphogenesis; Pathway interactions; Research; Research Personnel; Resources; Role; Source; United States National Institutes of Health; receptor

This subproject is one of many research subprojects utilizing the resources provided by a Center grant funded by NIH/NCRR. The subproject and investigator (PI) may have received primary funding from another NIH source, and thus could be represented in other CRISP entries. The institution listed is for the Center, which is not necessarily the institution for the investigator. Aim 1: Define the role of antipsychotics in dendrite morphogenesis. Aim 2: Define the downstream pathways in dendrite morphogenesis. Aim 3: Define the role of DHA and its metabolite, NPD1 in dendrite morphogenesis.

该副本是利用众多研究子项目之一 由NIH/NCRR资助的中心赠款提供的资源。子弹和 调查员（PI）可能已经从其他NIH来源获得了主要资金， 因此可以在其他清晰的条目中代表。列出的机构是 对于中心，这不一定是调查员的机构。 目标1：定义抗精神病药在树突形态发生中的作用。 目标2：定义树突形态发生中的下游途径。 AIM 3：定义DHA及其代谢产物NPD1在树突形态发生中的作用。