Renal Inflammation: Mechanisms and Consequences

肾脏炎症：机制和后果

• 批准号: 7500570
• 负责人: WILLIAM Evans MITCH
• 金额: $ 8.58万
• 依托单位: BAYLOR COLLEGE OF MEDICINE
• 依托单位国家: 美国
• 财政年份: 2007
• 资助国家: 美国
• 起止时间: 2007-05-01 至 2008-04-30
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/7500570
• 关键词: Acute; Adverse effects; Anti-Inflammatory Agents; Anti-inflammatory; Apoptosis; Attention; CCL2 gene; Cell Adhesion Molecules; Cell Line; Cell Proliferation; Cells; Chronic Kidney Failure; Cicatrix; Crescentic Glomerulonephritis; Data; Development; Disease; Doxycycline; End stage renal failure; Fibrosis; Future; Gene Transfer; Glomerular basement membrane antibody; Glomerulonephritis; Goals; Growth Factor; Immune; In Vitro; Infiltration; Inflammation; Inflammatory; Inflammatory Response; Injury; Intercellular adhesion molecule 1; Interleukin-1; Kidney; Kidney Diseases; Light; MHC Class II Genes; Mediating; Mediator of activation protein; Microbubbles; Modeling; NF-kappa B; Outcome; Outcome Study; Pathogenesis; Pathway interactions; Phosphorylation; Play; Prevention; Principal Investigator; Property; Protein Overexpression; Rattus; Regulation; Research; Research Personnel; Resolution; Role; Signal Pathway; Signal Transduction; Signaling Protein; Smad Proteins; Smad protein; Staging; System; T-Lymphocyte; TNFRSF5 gene; Testing; Therapeutic; Tubular formation; Ultrasonography; Work; base; chemokine; cytokine; fibrogenesis; in vivo; inhibitor/antagonist; insight; macrophage; novel; novel therapeutics; p65; programs; receptor