Mechanisms underlying novel pro-survival peptides in neuroprotection

新型促生存肽神经保护的机制

• 批准号: 356951
• 负责人: Feng Zhong-Ping
• 金额: $ 55.73万
• 依托单位: University of Toronto
• 依托单位国家: 加拿大
• 项目类别: Operating Grants
• 财政年份: 2016
• 资助国家: 加拿大
• 起止时间: 2016-10-01 至 2021-10-01
• 项目状态: 已结题
• 来源: https://webapps.cihr-irsc.gc.ca/decisions/p/project_details.html?applId=356951&lang=en
• 关键词: Calcium Biniding Protein; Caltubin; Cell Biology; Cell Culture; Cell Survival; Microtubule; Neuroprotection; Peptide Drug; Pharmacology; Protein-Protein Interaction

Brain cell death following injuries or stresses often lead to permanent disabilities; this is largely due to the poor abilities of brain cells to re-grow. In contrast, the adult nerve cells of Lymnaea stagnalis, a freshwater pond snail, show a greater abi

受伤或压力后的脑细胞死亡通常导致永久性残疾；这在很大程度上是由于脑细胞重新生长的能力不佳。相比之下，淡水池塘蜗牛的lymnaea stagnalis的成年神经细胞显示出更大的ABI