Active Subversion of Innate Immunity by Bacterial LysM Protein

细菌 LysM 蛋白主动颠覆先天免疫

• 批准号: 8298307
• 负责人: Laurel L Lenz
• 金额: $ 39.63万
• 依托单位: NATIONAL JEWISH HEALTH
• 依托单位国家: 美国
• 财政年份: 2011
• 资助国家: 美国
• 起止时间: 2011-08-01 至 2012-02-14
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/8298307
• 关键词: Activated Natural Killer Cell; Animals; Anti-Bacterial Agents; Autoimmunity; Bacteria; Bacterial Infections; Bacterial Proteins; Binding; Biological; Carbohydrates; Categories; Cell surface; Cells; Cessation of life; Clinical; Cultured Cells; Data; Dendritic Cells; Disease; Food Contamination; Hematopoietic; Hepatic; Hepatocyte; Host resistance; Human; Immune; Immune response; Immunity; Incidence; Infection; Integration Host Factors; Interferon Type I; Interferon Type II; Interferons; Interleukin-1; Interleukin-10; Interleukin-18; Lead; Length; Ligands; Listeria monocytogenes; Maps; Membrane Glycoproteins; Modeling; Mus; N-terminal; NK Cell Activation; National Institute of Allergy and Infectious Disease; Natural Immunity; Natural Killer Cells; Pathogenesis; Pathogenicity; Peptides; Play; Population; Predisposition; Production; Protein Region; Proteins; Publishing; Role; SH3 Domains; Serum; Systemic infection; Testing; Tissues; Virulence Factors; Virulent; Virus Diseases; Work; base; cytokine; in vivo; macrophage; molecular imaging; mortality; mouse model; novel; pathogen; pathogenic bacteria; plant fungi; prevent; protective effect; receptor; recombinant peptide; response; tumor