Lung-Liver Axis During Pneumonia

肺炎期间的肺肝轴

• 批准号: 8076424
• 负责人: Lee Quinton
• 金额: $ 24.9万
• 依托单位: BOSTON UNIVERSITY MEDICAL CAMPUS
• 依托单位国家: 美国
• 财政年份: 2010
• 资助国家: 美国
• 起止时间: 2010-09-01 至 2013-06-30
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/8076424
• 关键词: Accounting; Acute Lung Injury; Acute-Phase Proteins; Acute-Phase Reaction; Address; Alveolar; Amyloid; Apoptotic; Bacterial Pneumonia; Binding; Biological; Biological Markers; Biological Process; Blood; C-reactive protein; Cell Line; Cells; Cessation of life; Chemotaxis; Complement; Cytokine Signaling; Data; Disease Progression; Edema; Environment; Escherichia; Escherichia coli; Gene Deletion; Gene Expression; Genes; Goals; Hepatic; Hepatocyte; Host Defense; Immune response; In Vitro; Individual; Infection; Infection prevention; Inflammation; Inflammatory; Inflammatory Response; Interleukin-1; Interleukin-6; Laboratories; Liver; Lower respiratory tract structure; Lung; Measures; Mediating; Mediator of activation protein; Mentors; Messenger RNA; Mus; NF-kappa B; Natural Immunity; Neutrophil Infiltration; Outcome; Patients; Phagocytosis; Phase; Plasma; Plasma Proteins; Pneumonia; Prevention; Protein Binding; Proteins; Public Health Schools; Receptor Signaling; Recombinant Cytokines; Research; Role; STAT3 gene; Serum; Serum amyloid A protein; Signal Pathway; Signal Transduction; Small Interfering RNA; Stat3 protein; Streptococcus pneumoniae; System; Testing; Transcriptional Regulation; Tumor Necrosis Factor Receptor; Tumor Necrosis Factor-alpha; Tumor Necrosis Factors; base; burden of illness; career; career development; chromatin immunoprecipitation; cytokine; in vivo; innovation; knock-down; leukocyte activation; lipopolysaccharide-binding protein; loss of function; lung injury; microbial; neutralizing antibody; new therapeutic target; novel strategies; programs; promoter; protein expression; response; tool; transcription factor