LIPID PEROXIDATION AND ANTIOXIDANT MECHANISMS

脂质过氧化和抗氧化机制

• 批准号: 7605665
• 负责人: Ned Allen Porter
• 金额: $ 0.05万
• 依托单位: VANDERBILT UNIVERSITY
• 依托单位国家: 美国
• 财政年份: 2006
• 资助国家: 美国
• 起止时间: 2006-12-01 至 2007-09-16
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/7605665
• 关键词: Antioxidants; Computer Retrieval of Information on Scientific Projects Database; Development; Free Radicals; Funding; Grant; Institution; Lipid Peroxidation; Lipids; Lipoproteins; Low-Density Lipoproteins; Proteins; Research; Research Personnel; Resources; Source; United States National Institutes of Health; oxidation

This subproject is one of many research subprojects utilizing the resources provided by a Center grant funded by NIH/NCRR. The subproject and investigator (PI) may have received primary funding from another NIH source, and thus could be represented in other CRISP entries. The institution listed is for the Center, which is not necessarily the institution for the investigator. Development of arterial plaque results from oxidation of lipoproteins, particularly low-density lipoproteins. The object of this study is to determine the mechanisms governing the free radical oxidation of lipids and proteins in LDL (low-density lipoproteins)

该子项目是利用该技术的众多研究子项目之一 资源由 NIH/NCRR 资助的中心拨款提供。子项目和 研究者 (PI) 可能已从 NIH 的另一个来源获得主要资金， 因此可以在其他 CRISP 条目中表示。列出的机构是 对于中心来说，它不一定是研究者的机构。 动脉斑块的形成是脂蛋白，特别是低密度脂蛋白氧化的结果。本研究的目的是确定控制 LDL（低密度脂蛋白）中脂质和蛋白质自由基氧化的机制