Prostate Cancer Parasitism of the HSC "niche" as a Molecular Mechanism for Ost

前列腺癌 HSC“生态位”的寄生作为 Ost 的分子机制

• 批准号: 8284222
• 负责人: RUSSELL S TAICHMAN
• 金额: $ 22.01万
• 依托单位: UNIVERSITY OF MICHIGAN AT ANN ARBOR
• 依托单位国家: 美国
• 财政年份: 2004
• 资助国家: 美国
• 起止时间: 2004-06-05 至
• 项目状态: 未结题
• 来源: https://reporter.nih.gov/project-details/8284222
• 关键词: ANXA2 gene; Accounting; Adhesions; Angiogenic Switch; Animals; Antibodies; Area; Binding; Biological Assay; Biology; Bone Marrow; Bone Marrow Transplantation; CXC Chemokines; CXCR4 Receptors; CXCR4 gene; Cells; Data; Endothelial Cells; Engraftment; Event; Foundations; Funding; Growth; Hematopoietic stem cells; Home environment; Homing; Homing Behavior; Implant; Indium; Invaded; Investigation; Ligands; Link; Location; Malignant Neoplasms; Malignant neoplasm of prostate; Marrow; Metastatic Neoplasm to the Bone; Metastatic Prostate Cancer; Modeling; Molecular; Neoplasm Metastasis; Osteoblasts; Pathway interactions; Peptides; Pisum sativum; Play; Primary Neoplasm; Prostate; Receptor Signaling; Role; Route; Signal Pathway; Signal Transduction; Skeleton; Stromal Cell-Derived Factor 1; Testing; Therapeutic Intervention; Time; Tissues; Work; bone; cancer cell; cell growth; foot; in vivo; metastatic process; neoplastic cell; novel; parasitism; receptor; stem cell niche; tumor