Role of Ca2+/calmodulin kinases in skeletal muscle glucose transport and growth.

Ca2/钙调蛋白激酶在骨骼肌葡萄糖转运和生长中的作用。

• 批准号: 8206323
• 负责人: Carol Ann Witczak
• 金额: $ 24.9万
• 依托单位: EAST CAROLINA UNIVERSITY
• 依托单位国家: 美国
• 财政年份: 2011
• 资助国家: 美国
• 起止时间: 2011-02-01 至 2014-01-31
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/8206323
• 关键词: 5' -AMP-activated protein kinase; Acquired Immunodeficiency Syndrome; Address; Affect; Aging; Atrophic; Attenuated; Biochemical; Body Weight decreased; Ca(2+)-Calmodulin Dependent Protein Kinase; Cachexia; Calcium/calmodulin-dependent protein kinase; Chronic lung disease; Complex; Data; Denervation; Development; Diabetes Mellitus; Disease; Equilibrium; Exhibits; Fatty acid glycerol esters; Functional disorder; Gene Transfer; Genetic Transcription; Goals; Growth; Heart Diseases; Hyperglycemia; Hypertrophy; Individual; Instruction; Kidney Diseases; Knockout Mice; Lead; Lipids; Malignant Neoplasms; Mammalian Cell; Metabolic; Metabolism; Modeling; Molecular; Muscle; Muscle Fibers; Muscle Proteins; Muscle Weakness; Muscular Atrophy; Patients; Pharmacologic Substance; Phase; Phosphotransferases; Physiological; Play; Process; Progress Reports; Protein Biosynthesis; Protein-Serine-Threonine Kinases; Proteins; Regulation; Role; Signal Pathway; Signal Transduction; Signaling Protein; Skeletal Muscle; Syndrome; Weight; Work; glucose transport; glucose uptake; in vivo; innovation; mTOR protein; mortality; muscle form; muscle hypertrophy; muscle metabolism; novel; oxidation; protein degradation; protein expression; response; skeletal muscle growth; skeletal muscle wasting; wasting