The Glucocorticoid Receptor as Signal Integrator: Studying All Drug Resistance

糖皮质激素受体作为信号积分器：研究所有耐药性

• 批准号: 8278345
• 负责人: MILES A PUFALL
• 金额: $ 24.15万
• 依托单位: UNIVERSITY OF IOWA
• 依托单位国家: 美国
• 财政年份: 2011
• 资助国家: 美国
• 起止时间: 2011-07-08 至 2014-06-30
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/8278345
• 关键词: Acute Lymphocytic Leukemia; Adverse effects; Allosteric Regulation; Apoptosis; Apoptotic; Asthma; Autoimmune Diseases; Award; B-Cell Acute Lymphoblastic Leukemia; Binding Sites; Biochemical; Biology; Calorimetry; Cell Line; Cells; Characteristics; Childhood; Chromatin; Communication; Complex; Computing Methodologies; Cues; DNA; DNA Binding; DNA Binding Domain; DNA Sequence; Data Set; Development; Diabetes Mellitus; Disease; Drug resistance; Drug usage; Face; Future; Gene Expression; Genes; Genetic Transcription; Genome; Genomics; Glucocorticoid Receptor; Glucocorticoid-induced apoptosis; Glucocorticoids; Goals; Health; Hematopoietic; Histone H3; Hormones; Hypertension; In Vitro; Intervention; Learning; Lysine; Malignant Neoplasms; Maps; Mass Spectrum Analysis; Measures; Messenger RNA; Methods; Modeling; Modification; Nuclear Extract; Nuclear Magnetic Resonance; Organ Transplantation; Osteoporosis; Outcome; Output; Pharmaceutical Preparations; Pre-B Acute Lymphoblastic Leukemia; Prednisone; Property; Proteins; RNA; Reading; Resistance; Resources; Response Elements; Screening procedure; Signal Pathway; Signal Transduction; Specific qualifier value; Structural Models; System; Testing; Thermodynamics; Time; Tissues; Training; Translating; Work; abstracting; base; career development; cell type; effective therapy; experience; genome-wide; glucocorticoid-induced orphan receptor; hormone resistance; human tissue; outcome forecast; programs; receptor; receptor binding; receptor function; response; transcription factor