ROS Signaling in Baroreeeptor and Sympathetic Neurons in Normal and Heart Failure

正常和心力衰竭压力感受器和交感神经元中的 ROS 信号转导

• 批准号: 8154145
• 负责人: MARK W CHAPLEAU
• 金额: $ 51.43万
• 依托单位: UNIVERSITY OF IOWA
• 依托单位国家: 美国
• 财政年份: 2010
• 资助国家: 美国
• 起止时间: 2010-07-15 至 2013-06-30
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/8154145
• 关键词: Address; Age; Angiotensin II; Angiotensinogen; Animals; Antioxidants; Apolipoprotein E; Arrhythmia; Atherosclerosis; Baroreflex; Blood Pressure; Calcium/calmodulin-dependent protein kinase; Cardiovascular Physiology; Cause of Death; Coronary; Development; Efferent Neurons; Endothelin; Exhibits; Experimental Models; Failure; Functional disorder; Gene Transfer; Goals; Heart failure; Human; Hyperlipidemia; Hypertension; Infarction; Injection of therapeutic agent; Intervention; Ion Channel; Left Ventricular Dysfunction; Ligation; Mediating; Methionine; Modeling; Mus; NADPH Oxidase; Nerve; Neurons; Oxidative Stress; Oxides; Oxidoreductase; Patients; Peptides; Physiological; Pressoreceptors; Production; Protein Isoforms; Protein Kinase C; Reactive Oxygen Species; Renin; Role; Severities; Signal Pathway; Signal Transduction; Transgenic Mice; Viral; blood pressure regulation; cardiovascular risk factor; clinically relevant; disability; hypercholesterolemia; improved; in vivo; inhibitor/antagonist; insight; mortality; mouse model; neuronal excitability; novel; receptor; response; restoration