Basophils in Modulating Th2 Responses in Human Allergic Disease

嗜碱性粒细胞调节人类过敏性疾病中的 Th2 反应

• 批准号: 8308732
• 负责人: JOHN T. SCHROEDER
• 金额: $ 41万
• 依托单位: JOHNS HOPKINS UNIVERSITY
• 依托单位国家: 美国
• 财政年份: 2011
• 资助国家: 美国
• 起止时间: 2011-08-15 至 2013-07-31
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/8308732
• 关键词: Address; Affect; Allergens; Allergic; Allergic Disease; Allergic inflammation; Ancillary Study; Antigen-Presenting Cells; Area; Asthma; Attention; Basophils; Blood; Blood Circulation; CD4 Positive T Lymphocytes; CD80 gene; CD8B1 gene; Cell Communication; Cell Maturation; Cells; Child; Cytoplasmic Granules; Data; Dendritic Cells; Development; Disease; Food; Generations; Genetic; Histamine; Histocompatibility Antigens Class II; Human; Hypersensitivity; IgE; Immune; Incidence; Inflammation; Inflammation Mediators; Interferons; Interleukin-13; Interleukin-3; Interleukin-4; Investigation; Laboratories; Lead; Leukocytes; Leukotriene C4; Ligands; Lung; MHC Class II Genes; Mediating; Mediator of activation protein; Memory; Modeling; Molecular; Mus; Pathogenesis; Phase; Phenotype; Play; Probability; Process; Production; Publishing; Reporting; Rest; Risk Factors; Role; Site; Source; Stimulus; Symptoms; System; T cell response; T-Cell Activation; T-Cell Proliferation; T-Cell Receptor; T-Lymphocyte; TLR2 gene; TLR7 gene; TSLP gene; Therapeutic Intervention; Tissues; Translating; Translations; United States; Work; atopy; clinically relevant; cytokine; granulocyte; in vivo; innovation; light (biology); memory CD4 T lymphocyte; mouse model; new therapeutic target; novel; novel therapeutics; research study; response