Occludin-specific HCV cell entry mechanisms

Occludin 特异性 HCV 细胞进入机制

• 批准号: 8337067
• 负责人: Matthew J Evans
• 金额: $ 42.13万
• 依托单位: ICAHN SCHOOL OF MEDICINE AT MOUNT SINAI
• 依托单位国家: 美国
• 财政年份: 2011
• 资助国家: 美国
• 起止时间: 2011-09-30 至 2012-09-29
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/8337067
• 关键词: Adverse effects; Amino Acids; Animal Model; Antibody Affinity; Antiviral Agents; Antiviral Therapy; Biological Models; CD81 gene; Cell physiology; Cells; Characteristics; Clathrin; Development; Endocytosis; Endosomes; Epitopes; Event; Generations; Genetic; Genotype; Glycoproteins; Glycosaminoglycans; Goals; Hepatitis C; Hepatitis C virus; Hepatocyte; Human; Individual; Interferons; Knowledge; Life Cycle Stages; Light; Liver diseases; Location; Low Density Lipoprotein Receptor; Mammalian Cell; Maps; Mediating; Methods; Mus; Mutagenesis; Mutation; Pan Genus; Peptides; Play; Process; Proteins; Public Health; Regulation; Research; Research Personnel; Role; SR-BI receptor; Surveys; System; Tight Junctions; Tissues; Tropism; United States; Variant; Viral; Viral Proteins; Virion; Virus; Virus Receptors; Work; base; cell type; claudin-1 protein; drug development; extracellular; human PHEMX protein; inhibitor/antagonist; innovation; insight; interest; mutant; novel; occludin; pathogen; protein protein interaction; receptor; research and development; research study; uptake