CORE--GENE EXPRESSION AND GENOMICS

核心——基因表达和基因组学

• 批准号: 7335644
• 负责人: ANDREW B. LEITER
• 金额: $ 18.76万
• 依托单位: TUFTS MEDICAL CENTER
• 依托单位国家: 美国
• 财政年份: 2006
• 资助国家: 美国
• 起止时间: 2006-12-01 至 2007-11-30
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/7335644
• 关键词: 1-Phosphatidylinositol 3-Kinase; Adenoviruses; Agonist; Antibodies; Arts; BHLH Protein; Basic Science; Bioinformatics; Biological; Boston; Cardiology; Cell Cycle Progression; Cell Cycle Regulation; Cholecystokinin; Cholecystokinin B Receptor; Cloning; Communities; Computer software; Consult; Consultations; Core Facility; Cultured Cells; Cyclic AMP Response Element; Data; Detection; Development; Digestive System Disorders; Drops; Drosophila genus; Endocrine; Ensure; Equipment; Fostering; Funding; Gastrointestinal tract structure; Gene Expression; Gene Expression Regulation; General Hospitals; Generations; Genetic; Genomics; Goals; Histology; Human Resources; Individual; Inflammatory Bowel Diseases; Knock-out; Laboratories; Licensing; Liver Regeneration; Massachusetts; Microarray Analysis; Microinjections; Modeling; Molecular; Molecular Biology; Mus; National Institute of Diabetes and Digestive and Kidney Diseases; Neurosecretory Systems; Numbers; Pancreatic Polypeptide; Parasites; Peptide YY; Peptides; Personal Satisfaction; Phosphoinositide-3-Kinase, Catalytic, Gamma Polypeptide; Polymerase Chain Reaction; Printing; Process; Production; Productivity; Protocols documentation; Publications; Purpose; Recombinants; Regulator Genes; Research; Research Institute; Research Personnel; Research Project Grants; Resources; Scanning; Scientific Advances and Accomplishments; Scientist; Secretin; Services; Signal Pathway; Spottings; Support of Research; System; Technical Expertise; Techniques; Technology; Time; Tissues; Training; Transcription Repressor/Corepressor; Transcriptional Regulation; Transgenic Mice; Transgenic Mouse Facility; Transgenic Organisms; Vendor; Virus; base; cellular imaging; cost; gastrointestinal; instrument; interest; member; novel; pathogenic bacteria; prosomatostatin; receptor; receptor function; tumorigenesis; vector

ABSTRACT NOT PROVIDED

未提供摘要