EFFECTS OF PARTIAL SLEEP DEP &/OR RECOVERY SLEEP ON SLEEPINESS&CYTOKINE SECRETI

部分睡眠 DEP 的影响

• 批准号: 7951232
• 负责人: ALEXANDROS N VGONTZAS
• 金额: $ 7.26万
• 依托单位: PENNSYLVANIA STATE UNIV HERSHEY MED CTR
• 依托单位国家: 美国
• 财政年份: 2009
• 资助国家: 美国
• 起止时间: 2009-04-01 至 2010-03-31
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/7951232
• 关键词: Clinical Research; Computer Retrieval of Information on Scientific Projects Database; Funding; Grant; Inflammatory; Institution; Interleukin-6; Mediator of activation protein; Pathology; Recovery; Research; Research Personnel; Resources; Sleep; Sleep Apnea Syndromes; Source; United States National Institutes of Health; cytokine

This subproject is one of many research subprojects utilizing the resources provided by a Center grant funded by NIH/NCRR. The subproject and investigator (PI) may have received primary funding from another NIH source, and thus could be represented in other CRISP entries. The institution listed is for the Center, which is not necessarily the institution for the investigator. The primary hypothesis is that pro-inflammatory cytokines IL-6 and TNFa are mediators of sleepiness and contribute to the pathology of sleep apnea.

该副本是利用众多研究子项目之一 由NIH/NCRR资助的中心赠款提供的资源。子弹和 调查员（PI）可能已经从其他NIH来源获得了主要资金， 因此可以在其他清晰的条目中代表。列出的机构是 对于中心，这不一定是调查员的机构。 主要假设是促炎性细胞因子IL-6和TNFA是嗜睡的介体，并有助于睡眠呼吸暂停的病理学。