Immunopathogenesis of HIV-1 Infection: Role of Methamphetamine

HIV-1 感染的免疫发病机制：甲基苯丙胺的作用

• 批准号: 7437563
• 负责人: MADHAVAN P. NAIR
• 金额: $ 7.72万
• 依托单位: FLORIDA INTERNATIONAL UNIVERSITY
• 依托单位国家: 美国
• 财政年份: 2006
• 资助国家: 美国
• 起止时间: 2006-04-01 至 2011-08-31
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/7437563
• 关键词: 1-Phosphatidylinositol 3-Kinase; AIDS Dementia Complex; Abbreviations; Adam11 gene; Affect; Antigen Presentation; Antigen-Presenting Cells; Antigens; Biological Models; CCL22 gene; CCL3 gene; CCL4 gene; CCR5 gene; CD209 gene; CD4 Antigens; CD4 Lymphocyte Count; CD4 Positive T Lymphocytes; CD40 Antigens; CD80 gene; CXCL12 gene; CXCR4 gene; Catabolism; Cell Proliferation; Cell surface; Cells; Dendritic Cells; Dioxygenases; Disease Progression; Dopamine; Dopamine D1 Receptor; Drug abuse; Enzymes; Essential Amino Acids; Face; Figs - dietary; Functional disorder; Gene Expression; Growth Factor; HIV Receptors; HIV-1; Immune response; Immune system; Immunity; Immunologic Deficiency Syndromes; Immunosuppressive Agents; In Vitro; Infection; Inositol; Kynurenine; Lead; Lymphocyte antigen CD50; Lymphoid Tissue; MAPK1 gene; MAPK14 gene; MAPK3 gene; MHC Class I Genes; Macrophage Inflammatory Proteins; Mediating; Methamphetamine; Microbe; Mitogen-Activated Protein Kinases; Molecular; Neuraxis; Neurotoxins; Numbers; Pathway interactions; Patients; Peptides; Peripheral Blood Mononuclear Cell; Persons; Phosphoinositide-3-Kinase, Catalytic, Gamma Polypeptide; Phosphotransferases; Play; Printing; Process; Production; Protein Kinase; Proteins; Quinolinic Acid; Quinolinic Acids; RANTES; Rate; Reporting; Research; Research Project Grants; Rest; Role; Signal Transduction; Signal Transduction Pathway; Small Inducible Cytokine A3; Small Interfering RNA; Stromal Cell-Derived Factor 1; T-Cell Activation; T-Lymphocyte; TNFRSF5 gene; Tryptophan; Up-Regulation; Viral Load result; Virus; Virus Diseases; Work; antigen processing; base; cell mediated immune response; chemokine; chemokine receptor; chlorambucil/dactinomycin/methotrexate protocol; drug of abuse; enzyme pathway; genetic variant; in vivo; indolamine; inhibitor/antagonist; methyl tryptophan; monocyte; prevent; receptor; research study; response; stress activated protein kinase; stress-activated protein kinase 1; tissue processing

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