Prostate Cancer and Obesogenic Factors

前列腺癌与肥胖因素

• 批准号: 9457412
• 负责人: Everardo Macias
• 金额: $ 21.78万
• 依托单位: DUKE UNIVERSITY
• 依托单位国家: 美国
• 财政年份: 2017
• 资助国家: 美国
• 起止时间: 2017-03-20 至 2019-06-30
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/9457412
• 关键词: Ablation; African American; Agonist; Androgen Receptor; Binding; Biological Process; Cancer Biology; Caucasians; Cell Cycle; Cell Cycle Arrest; Cell Death; Cell Proliferation; Cells; Cessation of life; ChIP-seq; Chronic; Cleaved cell; Coupled; Cyclic AMP-Dependent Protein Kinases; DNA; DNA Binding Domain; DNA-Protein Interaction; Dipeptidyl-Peptidase IV; ERG gene; EWSR1 gene; Event; Gene Fusion; Genes; Genome; Genomics; Gleason Grade for Prostate Cancer; Growth; Health; Helix-Turn-Helix Motifs; High Fat Diet; In Vitro; Integration Host Factors; Ligands; Link; Malignant Neoplasms; Malignant neoplasm of prostate; Mediating; Metabolic syndrome; Molecular; Mus; Mutation; Neoplasm Metastasis; Obese Mice; Obesity; Oncogenes; Open Reading Frames; Outcome; Peptides; Pharmacology; Phosphotransferases; Prostate; Protein-Serine-Threonine Kinases; Proteins; Radical Prostatectomy; Reading Frames; Recombinants; Reporting; Ribosomes; Risk Factors; Role; Signal Transduction; TMPRSS2 gene; Testing; Thinness; Tissues; Translations; Up-Regulation; VCaP; Winged Helix; Xenograft procedure; cancer cell; cancer recurrence; cancer stem cell; cancer type; fusion gene; healthy weight; high risk; inhibitor/antagonist; knock-down; lipid biosynthesis; melanoma; men; migration; mutant; neuropeptide Y; neuropeptide Y2 receptor; obesogenic; overexpression; promoter; proneuropeptide Y; prostate cancer cell; prostate cancer progression; prostate cancer risk; ribosome profiling; small hairpin RNA; stem; stem-like cell; transcription factor; transcriptome; tumor; tumor growth; tumorigenic; Ablation; African American; Agonist; Androgen Receptor; Binding; Biological Process; Cancer Biology; Caucasians; Cell Cycle; Cell Cycle Arrest; Cell Death; Cell Proliferation; Cells; Cessation of life; ChIP-seq; Chronic; Cleaved cell; Coupled; Cyclic AMP-Dependent Protein Kinases; DNA; DNA Binding Domain; DNA-Protein Interaction; Dipeptidyl-Peptidase IV; ERG gene; EWSR1 gene; Event; Gene Fusion; Genes; Genome; Genomics; Gleason Grade for Prostate Cancer; Growth; Health; Helix-Turn-Helix Motifs; High Fat Diet; In Vitro; Integration Host Factors; Ligands; Link; Malignant Neoplasms; Malignant neoplasm of prostate; Mediating; Metabolic syndrome; Molecular; Mus; Mutation; Neoplasm Metastasis; Obese Mice; Obesity; Oncogenes; Open Reading Frames; Outcome; Peptides; Pharmacology; Phosphotransferases; Prostate; Protein-Serine-Threonine Kinases; Proteins; Radical Prostatectomy; Reading Frames; Recombinants; Reporting; Ribosomes; Risk Factors; Role; Signal Transduction; TMPRSS2 gene; Testing; Thinness; Tissues; Translations; Up-Regulation; VCaP; Winged Helix; Xenograft procedure; cancer cell; cancer recurrence; cancer stem cell; cancer type; fusion gene; healthy weight; high risk; inhibitor/antagonist; knock-down; lipid biosynthesis; melanoma; men; migration; mutant; neuropeptide Y; neuropeptide Y2 receptor; obesogenic; overexpression; promoter; proneuropeptide Y; prostate cancer cell; prostate cancer progression; prostate cancer risk; ribosome profiling; small hairpin RNA; stem; stem-like cell; transcription factor; transcriptome; tumor; tumor growth; tumorigenic

No abstract provided

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