Passive social media use, coping, and momentary stress in geospatial context: longitudinal effects on mental health and intermediate biological pathways in a racially diverse sample of adolescents

地理空间背景下的被动社交媒体使用、应对和瞬时压力：对不同种族青少年样本中心理健康和中间生物途径的纵向影响

• 批准号: 10815348
• 负责人: DAVID LEE
• 金额: $ 67.77万
• 依托单位: OHIO STATE UNIVERSITY
• 依托单位国家: 美国
• 财政年份: 2023
• 资助国家: 美国
• 起止时间: 2023-09-14 至 2028-07-31
• 项目状态: 未结题
• 来源: https://reporter.nih.gov/project-details/10815348
• 关键词: Adolescent; Adult; Affect; Affective; Age; Area; Behavior; Biological; Biology; Black race; Blood; Blood Cells; C-reactive protein; Cellular Phone; Charge; Collection; Coping Skills; Crime; Cross-Sectional Studies; Data; Ecological momentary assessment; Emotional; Environment; Exposure to; Female; Frequencies; Gender; Gene Expression; Genes; Goals; Hormones; Hydrocortisone; Immune; Immune system; Individual Differences; Inflammation; Inflammatory; Interferons; Leukocytes; Life; Link; Location; Longitudinal Studies; Measurement; Measures; Mental Depression; Mental Health; Modeling; Monitor; Neighborhoods; Outcome; Parasympathetic Nervous System; Participant; Pathway interactions; Patient Self-Report; Personal Satisfaction; Persons; Phase; Physiological; Police; Predictive Factor; Probability; Process; Published Comment; Race; Records; Reporting; Research; Risk; Risk Factors; Role; Safety; Sampling; Signal Pathway; Signaling Protein; Stress; Stress and Coping; Stressful Event; Surveys; System; Teenagers; Telephone; Testing; Time; Viral; Work; Youth; biological adaptation to stress; complement C2a; coping; depressive symptoms; experience; girls; heart rate variability; inflammatory marker; male; model building; negative affect; novel; physical conditioning; psychobiology; psychologic; racial diversity; recruit; response; self esteem; social; social influence; social media; social stress; stressor; transcription factor; violent crime

Project Summary A concomitant rise in adolescent social media use and decline in adolescent mental health over the last decade has raised the question as to the degree of relationship between these factors. To better understand this relationship, we examine how passive social media use (pSMU)—monitoring other people’s lives without engaging in direct exchanges with others—leads to poor mental health (Aim 1), when people engage in pSMU and how this coping strategy impacts mental health (Aim 2), and for whom pSMU is likely to undermine mental health (Aims 3) in a longitudinal study (n=400) in adolescents (ages 13-17; 50% Male/Female; 50% Black/White youth). There will be a 2-month focused study period. The initial and final 2 weeks of the 2-month study period will use ecological momentary assessment-based surveys (5 prompts a day) to measure pSMU, and to characterize affective responses to both pSMU and stress exposures the youth encounter in daily life. Objective pSMU will also be measured using an app installed on the youth’s phone in order to continuously measure pSMU over the entire 2-month intensive study period. We will measure stress exposures and responses in two novel ways. First, using GPS tracking we will determine the youth’s exposures to objectively stressful environments (i.e., high crime areas). Second, we will use GPS to trigger EMA prompts at locations youth reported as being stressful at baseline. The influences of these experiences on mental health trajectories (measured at weeks 0,2,4,6,8 and 20 will be assessed. Additionally, we focus on the relationship of pSMU to two physiological pathways that are responsive to social stress and influence risk for poor mental health: a) the parasympathetic nervous system, the function of which will be continuously measured using a self-charging wristband worn by the youth for the entire 2-month intensive study period to quantify shifts in heart-rate variability (HRV); b) and the expression of immune and other socially stress responsive gene pathways in blood cells sampled on week 0,2,6, and 8. This will provide the opportunity to determine the effects of pSMU on momentary affect, momentary HRV, immune related gene expression, a marker of general inflammation (CRP), and changes in depressive symptoms (Aim 1). We will also determine the role of stressor exposure (self-reported and GPS based) upon pSMU (Aim 2a) and the degree to which pSMU as a stress coping strategy moderates the effects of stressor exposure on affective response, HRV, stress related gene signaling pathways, inflammation, and mental health measures (Aim 2b). To examine individual differences in these effects, we will determine the degree to which gender is associated with increased pSMU (Aim 3a) and whether gender moderates responses to pSMU (Aim 3b). Finally, because our sample will be half Black youth, we will also determine if race moderates the relationship between GPS derived stress exposures (including GPS determined exposure to heavily policed areas or racially exclusive areas) and pSMU (Aim 3c).

项目概要 过去一段时间，青少年社交媒体使用率上升，青少年心理健康状况下降 为了更好地理解这些因素之间的关系程度，十年提出了一个问题。 为了研究这种关系，我们研究了被动社交媒体使用（pSMU）如何在不影响他人的情况下监控他人的生活。 与他人直接交流——当人们参与 pSMU 时，会导致心理健康状况不佳（目标 1） 以及这种应对策略如何影响心理健康（目标 2），以及 pSMU 可能会损害哪些人的心理健康 青少年（13-17 岁；50% 男性/女性；50%）的纵向研究（n=400）中的健康（目标 3） 黑人/白人青年）将有为期 2 个月的重点学习期。这 2 个月的最初和最后 2 周。 研究期间将使用基于生态瞬时评估的调查（每天 5 个提示）来测量 pSMU， 并描述青少年在日常生活中遇到的 pSMU 和压力暴露的情感反应。 目标 pSMU 还将使用安装在青少年手机上的应用程序进行测量，以便持续 在整个 2 个月的强化研究期间测量 pSMU 我们将测量压力暴露和。 首先，我们将使用 GPS 跟踪来客观地确定青少年的暴露程度。 其次，我们将使用 GPS 在地点触发 EMA 提示。 青少年报告称在基线时有压力这些经历对心理健康轨迹的影响。 （将评估第 0、2、4、6、8 和 20 周测量的值。此外，我们重点关注 pSMU 与 对社会压力做出反应并影响心理健康不良风险的两种生理途径： 副交感神经系统，其功能将使用自充电持续测量 青少年在整个两个月的强化研究期间佩戴腕带，以量化心率变化 变异性（HRV）；以及免疫和其他社会应激反应基因途径的表达； 在第 0、2、6 和 8 周取样的血细胞。这将提供确定 pSMU 效果的机会 瞬时影响、瞬时 HRV、免疫相关基因表达、一般炎症标志物 （CRP）和抑郁症状的变化（目标 1）我们还将确定压力源暴露的作用。 （自我报告和基于 GPS）基于 pSMU（目标 2a）以及 pSMU 作为压力应对的程度 策略调节压力源暴露对情感反应、HRV、压力相关基因信号传导的影响 途径、炎症和心理健康指标（目标 2b）。 影响，我们将确定性别与 pSMU 增加的相关程度（目标 3a）以及 性别是否会调节对 pSMU 的反应（目标 3b）。最后，因为我们的样本将有一半是黑人青年， 我们还将确定种族是否会调节 GPS 衍生的压力暴露之间的关系（包括 GPS 确定暴露于戒备森严的区域或种族排斥区域）和 pSMU（目标 3c）。