MECHANISM OF REPLICATION TERMINATION

复制终止机制

• 批准号: 8171240
• 负责人: DEEPAK BASTIA
• 金额: $ 0.24万
• 依托单位: UNIVERSITY OF WASHINGTON
• 依托单位国家: 美国
• 财政年份: 2010
• 资助国家: 美国
• 起止时间: 2010-09-01 至 2011-08-31
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/8171240
• 关键词: Computer Retrieval of Information on Scientific Projects Database; DNA biosynthesis; Funding; Genetic Recombination; Genome Stability; Grant; Institution; Process; Regulation; Research; Research Personnel; Resources; Source; United States National Institutes of Health; clinically significant

This subproject is one of many research subprojects utilizing the resources provided by a Center grant funded by NIH/NCRR. The subproject and investigator (PI) may have received primary funding from another NIH source, and thus could be represented in other CRISP entries. The institution listed is for the Center, which is not necessarily the institution for the investigator. DNA replication is an essential process and its regulation has fundamental and clinical significance. In this context Replication termination is highly important because it regulates replication, connects it with recombination and confirms genome stability.

该子项目是利用该技术的众多研究子项目之一 资源由 NIH/NCRR 资助的中心拨款提供。子项目和 研究者 (PI) 可能已从 NIH 的另一个来源获得主要资金， 因此可以在其他 CRISP 条目中表示。列出的机构是 中心，不一定是研究者的机构。 DNA复制是一个重要的过程，其调控具有基础和临床意义。在这种情况下，复制终止非常重要，因为它调节复制，将其与重组联系起来并确认基因组稳定性。