Innate Immunity and Oral Carcinogenesis

先天免疫与口腔癌发生

• 批准号: 8287840
• 负责人: Ge Jin
• 金额: $ 35.33万
• 依托单位: CASE WESTERN RESERVE UNIVERSITY
• 依托单位国家: 美国
• 财政年份: 2011
• 资助国家: 美国
• 起止时间: 2011-07-15 至 2013-06-30
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/8287840
• 关键词: Ablation; Biological Assay; Biological Markers; C57BL/6 Mouse; CCL2 gene; Carcinoma in Situ; Cell Proliferation; Cell physiology; Cells; Data; Defensins; Development; Distal; Drug Delivery Systems; Fluorescence; Goals; Head and Neck Squamous Cell Carcinoma; Human; Immune; Immune response; Immune system; Immunity; Immunocompetent; Immunosuppressive Agents; In Situ Lesion; In Vitro; Incidence; Inflammation; Knockout Mice; Label; Luciferases; Lymphocyte; Malignant - descriptor; Malignant Neoplasms; Migration Assay; Molecular; Monocyte Chemoattractant Protein-1; Mouth Carcinoma; Mouth Neoplasms; Mus; Myelogenous; Myeloid Cells; Natural Immunity; Natural Killer Cells; Neoplasm Metastasis; Nude Mice; Population; Process; Proteins; Recruitment Activity; Research; Role; Signal Pathway; Site; Suppressor-Effector T-Lymphocytes; T-Lymphocyte; Testing; Therapeutic; Transfection; Tumor Immunity; Tumor Promotion; Tumorigenicity; angiogenesis; antimicrobial peptide; base; cancer cell; cell motility; chemokine receptor; cytokine; in vivo; inhibitor/antagonist; macrophage; malignant mouth neoplasm; matrigel; migration; monocyte; mouse model; neoplastic cell; neutralizing antibody; novel; oral carcinogenesis; outcome forecast; overexpression; response; trafficking; transcription factor; treatment strategy; tumor; tumor growth; tumor initiation; tumor progression; tumor xenograft; tumorigenesis; Ablation; Biological Assay; Biological Markers; C57BL/6 Mouse; CCL2 gene; Carcinoma in Situ; Cell Proliferation; Cell physiology; Cells; Data; Defensins; Development; Distal; Drug Delivery Systems; Fluorescence; Goals; Head and Neck Squamous Cell Carcinoma; Human; Immune; Immune response; Immune system; Immunity; Immunocompetent; Immunosuppressive Agents; In Situ Lesion; In Vitro; Incidence; Inflammation; Knockout Mice; Label; Luciferases; Lymphocyte; Malignant - descriptor; Malignant Neoplasms; Migration Assay; Molecular; Monocyte Chemoattractant Protein-1; Mouth Carcinoma; Mouth Neoplasms; Mus; Myelogenous; Myeloid Cells; Natural Immunity; Natural Killer Cells; Neoplasm Metastasis; Nude Mice; Population; Process; Proteins; Recruitment Activity; Research; Role; Signal Pathway; Site; Suppressor-Effector T-Lymphocytes; T-Lymphocyte; Testing; Therapeutic; Transfection; Tumor Immunity; Tumor Promotion; Tumorigenicity; angiogenesis; antimicrobial peptide; base; cancer cell; cell motility; chemokine receptor; cytokine; in vivo; inhibitor/antagonist; macrophage; malignant mouth neoplasm; matrigel; migration; monocyte; mouse model; neoplastic cell; neutralizing antibody; novel; oral carcinogenesis; outcome forecast; overexpression; response; trafficking; transcription factor; treatment strategy; tumor; tumor growth; tumor initiation; tumor progression; tumor xenograft; tumorigenesis

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