Mechanisms of Osteoporosis in Crohn's Disease

克罗恩病骨质疏松的机制

• 批准号: 6975249
• 负责人: FRANCISCO A SYLVESTER
• 金额: $ 0.04万
• 依托单位: UNIVERSITY OF CONNECTICUT SCH OF MED/DNT
• 依托单位国家: 美国
• 财政年份: 2004
• 资助国家: 美国
• 起止时间: 2004-04-15 至 2005-03-31
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/6975249
• 关键词: Crohn' s disease; T lymphocyte; biological models; children; clinical research; genetically modified animals; human subject; interleukin 10; laboratory mouse; leukocyte activation /transformation; osteopenia; osteoporosis; pathologic process; patient oriented research; tumor necrosis factor alpha; Crohn' s disease; T lymphocyte; biological models; children; clinical research; genetically modified animals; human subject; interleukin 10; laboratory mouse; leukocyte activation /transformation; osteopenia; osteoporosis; pathologic process; patient oriented research; tumor necrosis factor alpha

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